Boston, MA (PRWEB) April 06, 2013
Doctors Health Press, a division of Lombardi Publishing Corporation and publisher of various natural health newsletters, books, and reports, including the popular online Doctors Health Press e-Bulletin, is reporting on a new study finding that benfotiamine, a form of vitamin B1, could help stop the progression of Alzheimer’s disease.
As Doctors Health Press e-Bulletin (http://www.doctorshealthpress.com/brain-function-articles/this-nutrient-erases-memory-destroying-plaque) notes, benfotiamine is a fat-soluble form of thiamine. It’s considered a potent antioxidant that has been used over the past decade to treat pain symptoms. In Germany, benfotiamine is actually licensed for use as a treatment for nerve pain caused by sciatica.
As the article “This Nutrient Erases Memory-Destroying Plaque” reports, benfotiamine is also useful in preserving brain health, and the medical community is excited about its performance when it comes to protecting memory function. It turns out benfotiamine is linked to Alzheimer’s prevention because of its involvement in glucose metabolism.
As the article explains, when brain cells need fuel, they metabolize glucose. Sometimes, for a variety of reasons, metabolism slows down in the brain. When this happens, it seems to open the door for Alzheimer’s disease to take hold. Or at least, this is what researchers at the Department of Neurology and Neuroscience at Weill Cornell Medical College think may be the case.
The Doctors Health Press e-Bulletin article states that the researchers have discovered that patients with Alzheimer’s are often deficient in thiamine. This thiamine deficiency seems to encourage plaque formation in the brain. People who have low levels of thiamine have also been found to have trouble with their memory. Reverse this vitamin shortage, however, and the opposite is likely to happen, the Cornell researchers reasoned.
Doctors Health Press e-Bulletin reports that when the theory was tested on mice, those treated with benfotiamine showed diminished levels of plaque and their memory problems had reversed. It could be that this form of B1 could offer a safe and effective way to reverse memory-destroying plaque in the brain, helping to stop the progression of Alzheimer’s.
As the article concludes, benfotiamine is generally considered safe, but readers should talk to their doctor if they’re thinking about trying the supplement. For those who can’t seem to absorb enough thiamine, benfotiamine may be a better option. It’s easier for the body to break down and utilize.
(SOURCE: Gibson, G.E., et al, “Abnormal thiamine-dependent processes in Alzheimer's Disease. Lessons from diabetes,” Mol Cell Neurosci. July 2013; 55: 17-25.)
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