Cancer Dramatically Increases (WHO and WHY); the CBCD Explains
Rochester, NY (PRWEB) February 06, 2014 -- The CBCD has learned that “The World Cancer Report, produced by the WHO’s specialized cancer agency, predicts new cancer cases will rise from an estimated 14 million in 2012 to 22 million annually within two decades (1).” In the same time period, cancer deaths are to rise from 8.2 million to 13 million annually (1).
The big question is “why?”
The reason according to Dr. Hanan Polansky’s discovery is the increase in number of latent viruses residing in most people.
Doctors and scientists believe that latent viruses are harmless, and are dangerous only when reactivated. That is, when they cause symptoms, such as infectious mononucleosis.
This is a misconception.
Health professionals may then ask “Why is this a misconception?” Or, more specifically, “How can a latent virus cause cancer?”
Latency is essential for the survival for the virus. Therefore, latency is widespread. “The natural history … strongly suggests that the virus would disappear or at least would be significantly less prevalent if it were not able to establish a latent, silent infection.” (See the Annual Review of Microbiology, from 2013) (2).
Doctors must begin to realize that during the latent phase, the virus is not dormant. It is still active.
During the latent phase, it continues to make viral proteins and replicate. For instance, researchers wrote that human ganglia, which were infected with the latent Varicella Zoster Virus, showed multiple VZV transcripts. “RT-PCR and in situ hybridization studies have identified multiple VZV transcripts in latently infected human ganglia. State-of-the-art multiplex PCR technology, capable of detecting all 68 annotated VZV gene transcripts, revealed transcription of at least 12 VZV genes during latency.” (See the journal Viruses, from September 4, 2013) (3).
In short, viruses produce their proteins while latent.
How can the production of viral proteins during latency cause cancer?
The following is a simplified explanation of the Theory of Microcompetition with Foreign DNA as described by Dr. Hanan Polansky.
Dr. Polansky discovered that foreign DNA fragments, called N-boxes, cause most major diseases. When the foreign N-boxes belong to a virus, microcompetition between the viral DNA and the human DNA can lead to the development of many major diseases, such as cancer, even when the virus is latent or the viral DNA is broken into pieces and cannot express proteins.
The Center recommends that doctors and other health professionals turn to Dr. Polansky’s book, “Microcompetition with Foreign DNA and the Origin of Chronic Disease” for a better understanding of the risks posed by latent viruses.
How is the rise in cancer related to latent viruses?
Latent viral infections are increasing. More people across the world are becoming infected. Studies have shown hepatitis, herpes viruses (such as the Epstein Barr Virus, herpes zoster, and HSV-1 and HSV-2), and HPV infection rates have all increased over time. For example, one study found that “Between 1996 and 2003 older people accounted for 3.7% and 4.3%, respectively, of all GUM clinic attendances. The rate of STIs in older people more than doubled in 2003 compared with 1996 (p<0.0001).” (See the Journal of Sexually Transmitted Diseases, from October 2003) (4).
Another study found that “The proportion of newly diagnosed genital herpes infections resulting from HSV-1 increased from 31% in 1993 to 78% in 2001 (P <0.001, linear trend P <0.001).” (See the journal Herpes, from April 2004) (5). In addition, “The incidence rate (of herpes zoster) increased from 363.21 to 481.92 per 100,000 person-year in 2005-2012.” (See the journal Sexually Transmitted Infections, from August 2008) (6).
Since the number of latent viruses is increasing, and since a latent virus is causing cancer, the increase in number of latent viral infections is the reason for the rise in cancer cases.
To learn more about Dr. Hanan Polansky’s research and the Theory of Microcompetition with Foreign DNA, visit:http://www.cbcd.net.
References:
(1) WHO: Cancer cases tipped to rise 57% in 20 years in imminent 'human disaster'. Published on February 4, 2014.
http://edition.cnn.com/2014/02/04/health/who-world-cancer-report/
(2) An inquiry into the molecular basis of HSV latency and reactivation. Published in 2013.
http://www.ncbi.nlm.nih.gov/pubmed/24024635
(3) Varicella zoster virus (VZV)-human neuron interaction. Published on September 4, 2013.
http://www.ncbi.nlm.nih.gov/pubmed/24008377
(4) Trends in sexually transmitted infections (other than HIV) in older people: analysis of data from an enhanced surveillance system. Published in August 2008.
http://www.ncbi.nlm.nih.gov/pubmed/?term=Trends+in+sexually+transmitted+infections+(other+than+HIV)+in+older+people%3A+analysis+of+data+from+an+enhanced+surveillance+system.
(5) Increasing proportion of herpes simplex virus type 1 as a cause of genital herpes infection in college students. Published in October 2003.
http://www.ncbi.nlm.nih.gov/pubmed/?term=Increasing+proportion+of+herpes+simplex+virus+type+1+as+a+cause+of+genital+herpes+infection+in+college+students.
(6) Temporal trends in incidence rates of herpes zoster among patients treated in primary care centers in Madrid (Spain), 2005-2012. Published on November 16, 2013.
http://www.ncbi.nlm.nih.gov/pubmed/?term=Temporal+trends+in+incidence+rates+of+herpes+zoster+among+patients+treated+in+primary+care+centers+in+Madrid+(Spain)%2C+2005-2012.
The CBCD is a research center recognized by the IRS as a 501(c)(3) non-for-profit organization. The mission of the CBCD is to advance the research on the biology of chronic diseases, and to accelerate the discovery of treatments.
The CBCD published the “Purple” book by Dr. Hanan Polansky. The book presents Dr. Polansky’s highly acclaimed scientific theory on the relationship between foreign DNA and the onset of chronic diseases. Dr. Polansky’s book is available as a free download from the CBCD website.
Hanan Polansky, Center for the Biology of Chronic Disease (CBCD), +1 (585) 250-9999, [email protected]
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